#whatIsEpilepsy
Hi friends i’m doctor chief drum today i will discuss about epilepsy in its management a scissor is a paroxysmal event characterized by abnormal excessive high percent crona’s discharge of cortical neuron activity epilepsy can be defined as a chronic seas of disorder are a group of disorders characterized by scissors that usually reoccur unpredictably in the
Absence of a consistent provoking factor epilepsy is not contagious it is not a mental illness or a cognitive disability that neurological dysfunction seen in epilepsy can begin at birth childhood adelson’s or even in adulthood classification partial seizures are simple scissors without impairment of consciousness with motor symptoms with especial century or so
Made of sensory symptoms with psychic symptoms complex scissors with impairment of consciousness simple project onset followed by impairment of consciousness impaired consciousness it onset secondly generalized partial onset evolving too generalized tonic-clonic scissors generalized scissors absence myoclonic colonic tonic tonic colonic tonic infantile spasms and
Classified scissors status epilepticus partial scissors common 80% patients simple partial scissors do not cause loss of consciousness signs and symptoms modern convulsive jerking chewing motions or lips making sensory and somatosensory paresthesias auras automatic sweaty flashing pupil dilation behavioral hallucinations dysphasia impaired consciousness or ear
Complex partial seizures impairment of consciousness purposeless behavior is common affected person may wander about aimlessly aggressive behavior violence automatism picking it close real auditory are all factory hallucinations generalized scissors affecting both hemispheres defused three types idiopathic epilepsies age-related genetic origin symptomatic epilepsies
A consequence of a known suspected underlying disorder of cns cryptogenic phillips’s disorder of a hidden course age-related absence ceases petty small alterations of consciousness absence lost in ten to thirty seconds stalling with ocular i blinking and loss in postural tone 100 or more jelly onset occurs from 3 to 16 years disappear by 40 years myoclonic sudden
Involuntary jerking of facial limp or trend muscles in rhythmic manner chronic sustained muscle contractions alternating with relaxations tonic sustained muscle stiffening tony colonic jian mal epilepsy sudden loss of consciousness tonic face patient become rigid and falls to the ground respiration is interrupted big artist lost about one minute colonic face rapid
Muscle jerking muscle flaccidity incontinence trunk biting tachycardia heavy salivation during post acute phase head ache confusion nausea drowsiness disorientation may last for hours atomic drop attacks sudden loss of postural tom patient falls to the ground occur primarily in children unclassified scissors new nettle status epilepticus caesar rapid with no
Recovery of consciousness between attacks causes high fever especially in infants drug use alcohol withdrawal near drowning or lack of oxygen from another cause metabolic disturbances head trauma brain tumor infection stroke complication of diabetes our pregnancy causes of epilepsy stroke brain tumor brain infection past head injury drug use alcohol withdrawal
Metabolic problems other neurological conditions high fever especially in infants genetic factors epilepsy may occur with developmental disabilities autism cognitive impairments but the majority of people who have epilepsy do not have other impairments in very normal lives prieto physiology of epilepsy peril of discharges in cortical neurons aziza originates from
Great measure of any cord occur or subcortical area that causes abnormal flowering of neurons in breakdown of normal membrane conductance and inhibitory synaptic crums that causes locally focal caesar in widely generalized season abnormality of potassium conductance defect in voltage-sensitive ion channels deficiency in membrane atp’s neuron membrane in stability
Causes seizures deficiency of in habit or neurotransmitters increase in excitatory neurotransmitters promotes abnormal neuronal activity that causes caesar diagnosis of epilepsy electroencephalogram eeg neurological imaging studies magnetic resonance imaging mri functional mri computed tomography ct scan positron emission tomography pet single photon emission
Computerized tomography spe ct you can see the chart showing the complete management of all types of epilepsy and charlie is also showing scissor type with drug of first choice second choice third choice in fourth choice general majors for epilepsy immediate emergency measures if patient is seen convulsing in short that patient does not harm himself moving person
Away from sharp objects clothing about the neck should be loosened ensure the airway is clear removed any secretions are vomitus from the mouth or nose do not force a spoon or tongue depressor into the mouth remove false teeth in present after convulsions seize turn the patient into semi prone position by turning the patient on the site with one leg bent in the
Other leg straight monitor fits for its chart pharmacological treatment of epilepsy anticonvulsant drug therapy traditionally a single caesar has been regarded is an indication for investigation and assessment but not for drug treatment unless a second attack follows closely are based on the circumstances of the season drug treatment should certainly be considered
After two caesars in the type of drug depends on the type of caesar the underlying abnormality must also be corrected impossible guidelines for use of antiepileptic drugs begin with a single drug a the lowest dosage rate if ceases not controlled increased those two upper limit of dosage range or until side effects appear if caesar’s are poorly controlled change to
A different drug by gradually reducing those of initial agent while simultaneously introducing the new one this usually takes three to four weeks try three single drugs before resorting to drug combinations which help in only minority of cases treatment can be stopped only after two years season three and after a full evolution in discussion with patient grand
Mal epilepsy gradually increasing doses of a single drug should be tried first tablet sodium valproate 250 to 500 milligram one tablet twice daily originally increased to a maximum of 16 milligram per kg birthday in three divided ptosis if not controlled tablet levy trots item 25 milligram one tablet deli 200 milligram five tablets daily in two to three divided
Doses if not control the interpret sodium valproate 500 milligram system milligram per kg per day in three divided tosses plus tablet lamotrigine 25 milligram half tablet once daily to 100 milligram five tablets in two divided doses or tablet sodium valproate 500 milligram 60 milligram per kg per day and three divided ausa’s plus tablet topiramate 50 milligram
One tablet daily for one week then one tablet twice daily maximum 16 tablets in two doses partial scissors visually increasing doses of a single drug should be tried first
Transcribed from video
EPILEPSY, SIGNS, SYMPTOMS, CAUSES, DIAGNOSIS AND MANAGEMENT By Dr Jeewat Ram Health \u0026 Beauty TV